Question special
Lead Moderator

What are the thoughts on this? Dr. Capodanno has raised an interesting point and observation: "In ACC/AHA lesions A or B1, the risk ratio was 3.05 (BRS 3-fold significantly worse than EES), while in lesions B2 or C it was 1.1 (BRS similar to EES). This sounds counterintuitive, as it apparently suggests that BRS behave better in more complex lesions and worse in simpler ones." What might be some of the mechanisms behind this? Or is it simply that more drastic effects might be seen with a more complex lesion as a result of normal physiological processes?